By Donald D. Tresch, Wilbert S. Aronow
Offers up to date wisdom in regards to the pathophysiology of cardiovascular problems and healing advances for treating aged cardiac sufferers. This moment version comprises new chapters and up to date details at the potency and adversarial reactions of familiar and newly built cardiovascular medications, the administration of atrial traumatic inflammation, directions for using atnicoagulation in aged sufferers, and extra.
Read Online or Download Cardiovascular Disease in the Elderly Patient (Fundamental and Clinical Cardiology, V. 36.) PDF
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Extra resources for Cardiovascular Disease in the Elderly Patient (Fundamental and Clinical Cardiology, V. 36.)
In 10% (51/490); mitral valve annulus in 39% (190/490)—very heavy deposits in 13% (63/490), and in the apices of 1 or both left ventricular papillary muscles in 17% (85/ 490). The cardiac calcific deposits were more frequent and heavier in the nonagenarians than in the octogenarians. Numbers of patients with narrowing of 1 or more major epicardial coronary arteries. Among the 490 patients, 194 (40%) had none of the three major (right, left anterior descending, and left circumflex) epicardial coronary arteries narrowed by plaque Ͼ75% in cross-sectional area; 89 (18%) had one artery so narrowed; 105 (21%) had two arteries so narrowed; and 94 (19%) had all three arteries so narrowed.
5 Morphological Features 21 Figure 1 Tortuous and heavily calcified coronary arteries in a 95-year-old woman (SH #A80-74) who never had symptoms of cardiac dysfunction and who died from a perforated gastric ulcer. Top left: postmortem radiogram of the excised right (R), left main (LM), left anterior descending (LAD), and left circumflex (LC) coronary arteries. Top center: radiogram of a portion of the heart after removing the walls of the atria and most of the walls of the ventricles. MVA ϭ mitral valve annulus.
Cardiac volumes, ejection fraction, and fractional shortening) (2,28,29). Similar to animal studies, echocardiographic left ventricular wall thicknesses and left ventricular mass increase with age, due most likely to an increase in afterload with increasing age (2). Also similar to animal models, there is a slowed and delayed left ventricular relaxation in healthy elderly subjects (30). Thus, noninvasive studies have demonstrated an age-associated decrease in early diastolic left ventricular filling (2,31,32).